Arterial hypoxemia and performance during intense exercise
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Arterial hypoxemia and performance during intense exercise

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Published .
Written in English

Subjects:

  • Anoxemia,
  • Oxygen in the body,
  • Exercise for men -- Physiological aspects,
  • Cycling -- Physiological aspects

Book details:

Edition Notes

Statementby Maria D. Koskolou.
The Physical Object
FormatMicroform
Paginationxi, 47 leaves
Number of Pages47
ID Numbers
Open LibraryOL13598281M
OCLC/WorldCa29247795

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Arterial hypoxemia and performance during intense exercise. Koskolou MD(1), McKenzie DC. Author information: (1)Department of Sport Science, Allan McGavin Sports Medicine Centre, University of British Columbia, Vancouver, by: Get this from a library! Arterial hypoxemia and performance during intense exercise. [Maria D Koskolou]. It is concluded that maximal performance capacity is significantly impaired in highly trained cyclists working under an %S aO2 level of 87% but not under a milder desaturation level of 90%. Abstract. Exercise-induced arterial hypoxemia (EIAH) is characterized by the decrease in arterial oxygen tension and oxyhemoglobin saturation during dynamic aerobic exercise. Since the time of the initial observations, our knowledge and understanding of EIAH has grown, but many unknowns remain. The purpose of this review is to provide an update on recent findings, highlight areas of disagreement, Author: Paolo B. Dominelli, A. William Sheel.

Exercise-induced arterial hypoxemia (EIAH) is characterized by the decrease in arterial oxygen tension and oxyhemoglobin saturation during dynamic aerobic exercise. This study examined the effect of running and cycling on exercise‐induced arterial hypoxemia (EIAH) in individuals well trained in each modality. Thirteen male triathletes (x̄±SD: age=36±5 years, mass=69±8 kg, body fat=12±1%) performed progressive exercise to exhaustion during Cited by: 8. 1 Institute for Exercise and Environmental Med. and The Univ. of Texas Southwestern Med. Ctr., Many endurance athletes, regardless of age or gender, appear to experience exercise-induced arterial hypoxemia (EIAH). The purpose of this symposium will be to address the current theories of EIAH and present new information adding to our. desaturation during exercise that was equal in both control and added dead space studies. The decrease in maximal O 2 consumption with added dead space suggests that maximal exercise in cystic fibrosis is limited by respiratory factors. We subsequently examined whether pulmonary mechanics or arterial hypoxemia limits maximal exercise by:

Exercise-induced arterial hypoxemia is unaffected by intense physical training: a case report. Paolo B. Dominelli, a Glen E. Foster, b Giulio S. Dominelli, c William R. Henderson, a d Michael S. Koehle, a e Donald C. McKenzie, a e A. William Sheel a * a School of Kinesiology, University of British Columbia, Vancouver, BC V6T 1Z3, by: 3.   In hypoxemic COPD patients, long-term oxygen therapy has been found to improve survival, exercise, sleep, and cognitive performance with a therapeutic goal of oxygen saturation level over 90% during rest, sleep, and exertion. 13 Patients with SEIH require monitoring while using ambulatory systems during physical activity, to assure adequate oxygenation. Patients with ongoing Cited by: 6. According to Dempsey and Wagner, arterial hypoxemia induced by exercise was defined as an arterial O 2 saturation exercise greater than 10%). Measurements of cardiac parameters (heart rate, stroke volume) and calculation of 𝑄̇, a-vO 2 diff. Sixteen healthy, active men were studied to determine the effects of severe arterial hypoxemia on the electrocardiograms during exercise. The electrocardiograms were all normal at maximum heart rate while the subject breathed ambient air. During maximal exercise breathing ten percent oxygen (mean arterial oxygen pressure [P02] of 31 mm Hg), only one of the 16 had ST segment changes suggestive Cited by: 4.